Dementia Action Collaborative 17 · In-home care to assist with bathing asthmatic bronchitis 15 order 100 mcg ventolin free shipping, dressing asthma treatment success rates purchase ventolin 100 mcg otc, getting ready in the morning or evening asthma 504 generic 100 mcg ventolin amex, other personal care tasks/routines asthma symptoms poster 100mcg ventolin sale. Action Steps See Action Steps on pages 7, 9, and 13 and do the following: q Request a home safety evaluation with a Physical or Occupational Therapist to make the home safer and home care tasks easier. Discuss and document how your loved one wants to live at the end of their life, including medical care wanted or not wanted, comfort measures, and palliative and hospice care. In addition to services that can help a person with dementia to stay at home, there are many different types of homes or facilities that provide long-term care. Residential care options in Washington state include adult family homes, assisted living facilities and nursing homes. Start here: · Community Living Connections (Area Agency on Aging) for your area may be found at 855-567-0252 or I wish we had reached out earlier when we could have benefitted from palliative care as well. In the late stage, your loved one is completely dependent for personal care activities. At the same time, you may notice that they are still attuned to the emotions of others, enjoy companionship, respond to physical touch or music, and can encourage others to slow down and focus on the present. Respect yourself for the love and support you have provided for your loved one and yourself. Dementia Action Collaborative 23 Communication tips Dementia damages pathways in the brain. Brain changes make it difficult for a person with dementia to say what they want and understand what others are saying. Your loved one may have trouble coming up with the right words or a name, or may invent new words. The resulting misunderstandings can fray nerves all around, making communication even more difficult. You can expect that over time, a person living with dementia may struggle to organize their message, lose their train of thought or speak less often. When speaking, turn your face towards them and make sure your face is in the light so they can easily see your lip movements. Slow pace of speech slightly and allow time for the person to process and respond. Gestures or other visual cues can help promote better understanding than words alone. Rather than asking if your loved one needs to use the toilet, walk them to the toilet and point to it. Try to avoid arguing-no one will "win" and it will only lead to embarrassment, frustration or anger. It can be helpful to talk with others in the same situation to get more ideas and support. I needed to always calm and center myself before each visit so that she would be calm too. Your loved one should have: - A Health Care Directive (also called a "living will" or "advance directive" regarding treatment preferences); and - A Durable Power of Attorney for Health Care, appointing a health care "agent. Important topics of discussion include: Encouraging a diagnosis; Discussing safety issues, such as driving and safe medication use; Needing support with financial or legal planning; Coordinating care at home; Considering safe living situation and options; Discussing ways to support the primary care partner/caregiver. The plan also called for the formation of a next generation workgroup to implement the recommendations. Once you have registered and/or logged in, put the title (Dementia Road Map) in the search box and click Enter 4. Click Proceed to Checkout and follow onsite instructions To request this publication in Spanish, email: inquirywa@alz. Autism Tool Kit for Dental Professionals these materials are the product of on-going activities of the Autism Speaks Autism Treatment Network, a funded program of Autism Speaks. Department of Health and Human Services, Health Resources and Services Administration, Maternal and Child Health Research Program to the Massachusetts General Hospital. This tool kit is meant to be used in conjunction with the Dental Guide developed by Autism Speaks Family Services Community Connections. The Dental Guide provides important information about oral health and dental hygiene techniques for families and is available at This tool kit for Dental Professionals may help providers feel successful with this special group of patients. Autism Defined the term Autism describes a brain disorder that affects social interaction, communication and often results in repetitive or stereotyped behavior. Autism may refer to a specific diagnosis that is consistent with a number of specified symptoms. Other individuals may exhibit delay in their acquisition of language skills or have qualitative differences in the ways in which they communicate. They may have trouble with "to and fro" conversation, for example, or may engage in stereotyped or repetitive speech. These behaviors can include repetitive body movements or using objects in a repetitive manner rather than using the objects in the ways in which they were intended to be used. They may have strong positive or negative reactions to sounds, smells, sights, taste, texture or human touch. What will be different is the flow of these patients through your office and management techniques that can be employed to have a successful visit. This may be due to a variety of factors Flattening of the teeth as a result of bruxism including behavioral difficulties that make oral hygiene at home difficult and a poor diet higher in fermentable carbohydrates and sugars. The entire office staff, from the receptionists to the dental assistants, can be educated on how to properly manage these patients and welcome them into your dental practice. Ideally, to reduce caries rates it is crucial to teach primary caregivers how to provide optimal home care. When children are unable to effectively communicate their feelings of anxiety, they may demonstrate noncompliant or uncooperative behavior. There are a number of behavioral and environmental techniques that may help alleviate anxiety and increase cooperation. We discuss ways to help reduce anxiety and increase compliance throughout this guide. Parents will be concerned about both the child having an unpleasant experience and about their own embarrassment in the event the child is non-compliant or has a behavioral outburst. The best solution is for the parent, the child and the dentist to meet and develop a plan ahead of the visit. The parent will want to know what to expect in order to prepare the child but will also want to identify any stumbling blocks that may need to be discussed and overcome. It will help parents to relax, too, once they understand that everyone in the office is supportive. The dentist will want to learn more about the child, including what behavioral strategies the parent has found to be successful with their child. Finally, the child will be more likely to have a successful visit if given the chance to see the office, meet the staff, and learn what to expect. The techniques outlined in this tool kit may help you and your patient have a successful visit. Please refer to the intake form in the Autism Speaks Family Services Community Connections Dental Guide included here in Appendix A on page 20. Consider scheduling the visit for a time when the office is less busy or after hours. Provide education around a home care plan for regular tooth-brushing and flossing. This may also include a plan for desensitizing the child at home to some of the procedures that will occur during routine visits. It is important to understand that autism is not a single condition, but rather a group of related condition with multiple causes. Genetic research has now identified over 30 autism risk genes, and in about 20% of cases, a specific genetic cause can be identified. In most cases, autism is the result of a combination of genetic and environmental risk factors, particularly factors influencing development during the prenatal period. Parents may experience guilt about their possible role in causing the disorder in their child, and they may feel compelled to try anything plausible that gives some hope of improvement in their child.
This means the disease seems to occur at random with no clearly associated risk factors and no family history of the disease asthmatic bronchitis or pneumonia discount ventolin 100 mcg otc. Interestingly asthma symptoms 3 months buy 100mcg ventolin, the same mutation can be associated with atrophy of frontal-temporal lobes of the brain causing frontal-temporal lobe dementia asthma treatment step up effective ventolin 100 mcg. Some of the early symptoms include: · fasciculations (muscle twitches) in the arm asthma treatment pdf buy 100 mcg ventolin mastercard, leg, shoulder, or tongue · muscle cramps · tight and stiff muscles (spasticity) · muscle weakness affecting an arm, a leg, neck or diaphragm. In other cases, symptoms initially affect one of the legs, and people experience awkwardness when walking or running or they notice that they are tripping or stumbling more often. Individuals may develop problems with moving, swallowing (dysphagia), speaking or forming words (dysarthria), and breathing (dyspnea). Although the sequence of emerging symptoms and the rate of disease progression vary from person to person, eventually individuals will not be able to stand or walk, get in or out of bed on their own, or use their hands and arms. A small percentage of individuals may experience problems with language or decision-making, and there is growing evidence that some may even develop a form of dementia over time. They eventually lose the ability to breathe on their own and must depend on a ventilator. Affected individuals also face an increased risk of pneumonia during later stages 5 of the disease. Other gene mutations indicate defects in the natural process in which malfunctioning proteins are broken down and used to build new ones, known as protein recycling. Still others point to possible defects in the structure and shape of motor neurons, as well as increased susceptibility to environmental toxins. This observation provides evidence for genetic ties between these two neurodegenerative disorders. Researchers are investigating a number of possible causes such as exposure to toxic or infectious agents, viruses, physical trauma, diet, and behavioral and occupational factors. However, the presence of upper and lower motor neuron symptoms strongly suggests the presence of the disease. However, they can reveal other problems that may be causing the symptoms, such as a spinal cord tumor, a herniated disk in the neck that compresses the spinal cord, syringomyelia (a cyst in the spinal cord), or cervical spondylosis (abnormal wear affecting the spine in the neck). However, there are treatments available that can help control symptoms, prevent unnecessary complications, and make living with the disease easier. Supportive care is best provided by multidisciplinary teams of health care professionals such as physicians; pharmacists; physical, occupational, and speech therapists; nutritionists; social workers; respiratory therapists and clinical psychologists; and home care and hospice nurses. These teams can design an individualized treatment plan and provide special equipment aimed at keeping people as mobile, comfortable, and independent as possible. Although it is not entirely understood how the drug works, riluzole is believed to reduce damage to motor neurons by decreasing levels of glutamate, which transports messages between nerve cells and motor neurons. Riluzole does not reverse the damage already done to motor neurons, and people taking the drug must be monitored for liver damage and other possible side effects. Drugs also are available to help individuals with pain, depression, sleep disturbances, and constipation. Gentle, low-impact aerobic exercise such as walking, swimming, and stationary bicycling can strengthen unaffected muscles, improve cardiovascular health, and help people fight fatigue and depression. Range of motion and stretching exercises can help prevent painful spasticity and shortening (contracture) of muscles. Occupational therapists can suggest devices such as ramps, braces, walkers, and wheelchairs that help individuals conserve energy and remain mobile. They can recommend aids such as computer-based speech synthesizers that use eye-tracking technology and can help people develop ways for responding to yes-or-no questions with their eyes or by other nonverbal means. These methods and devices help people communicate when they can no longer speak or produce vocal sounds. Nutritionists can teach individuals and caregivers how to plan and prepare small meals throughout the day that provide enough calories, fiber, and fluid and how to avoid foods that are difficult to swallow. When individuals can no longer get enough nourishment from eating, doctors may advise inserting a feeding tube into the stomach. The use of a feeding tube also reduces the risk of choking and pneumonia that can result from inhaling liquids into the lungs. Breathing support As the muscles responsible for breathing start to weaken, people may experience shortness of breath during physical activity and difficulty breathing at night or when lying down. There are several techniques to help people increase forceful coughing, including mechanical cough assist devices and breath stacking. In breath stacking, a person takes a series of small breaths without exhaling until the lungs are full, briefly holds the breath, and then expels the air with a cough. Doctors may place a breathing tube through the mouth or may surgically create a hole at the front of the neck and insert a tube leading to the windpipe (tracheostomy). People may choose to be fully informed about these considerations and the long-term effects of life without movement before they make decisions about ventilation support. Increasing evidence also suggests that various types of glial support cells and inflammation cells of the nervous system may play an important role in the disease. Epigenetic changes can switch genes on and off, and thus can profoundly affect the human condition in both health and disease. These changes can occur in response to multiple factors, including external or environmental conditions and events. Biomarkers Biomarkers are biological measures that help to identify the presence or rate of progression of a disease or the effectiveness of a therapeutic intervention. Biomarkers can be molecules derived from a bodily fluid (such as those in the blood and cerebrospinal fluid), an image of the brain or spinal cord, or a measure of the ability of a nerve or muscle to process electrical signals. This work involves tests of drug-like compounds, gene therapy approaches, antibodies, and cell-based therapies. Clinical trials Many neurological disorders do not have effective treatment options. Clinical trials offer hope for many people and an opportunity to help researchers find better ways to safely detect, treat, or prevent disease. S, that supply investigators with tissue from people with neurological and other disorders. The goal is to increase the availability of, and access to , high quality specimens for research to understand the neurological basis of the disease. No cure yet exists, with one approved medication appearing to slow the disease process. These patients are faced with important decisions as their condition worsens: Will they want nutritional support through a gastrostomy? For respiratory assistance, will their preference be noninvasive ventilation or mechanical ventilation via tracheostomy? Providers need to educate patients and their caregivers regarding the disease process and ensure that patients receive appropriate care to meet their needs and preferences. Although life may be prolonged by the one currently available pharmacologic agent, no treatment option is yet capable of stopping or reversing progression of the disease. In addition to symptom form, which can be autosomalmanagement, supportive mea- dominant or autosomal-recessures, including physical, occu- sive. Patients with pseudobulbar palsy may exhibit inappropriate, excessive yawning and emotional outbursts; these manifestations are referred to as emotional incontinence. This syndrome has been linked to a neurotoxin in the seed of the cycad nut, a tropical plant endemic to the area, which was used in the 1950s and 1960s in the human food supply. Patients may notice increased episodes of tripping, clumsiness when they run or walk, a "dropped foot" gait, and/ or a decline in manual dexterity. Occasionally, patients encounter bladder dysfunction (urgent micturition), sensory symptoms, and cognitive symptoms (eg, dementia, parkinsonism). They may complain of slurred speech, nasal or low-volume speech, and/or inhibited tongue mobility. Those with axial truncal weakness often complain of difficulty maintaining erect posture when standing and of stooping as they walk. Some patients support the trunk by placing their hands in their front pants pockets or on their upper thighs. As the disease progresses, focal wasting of muscle groups occurs in all four extremities. Particularly involved are the muscles of the hands, forearms, or shoulders in the upper limbs; and of the proximal thigh or distal foot muscles in the lower limbs. Tongue fasciculations will be present, as will atrophy and diminished mobility of the tongue. The patient may have a history of exaggerated expression of emotion, such as uncontrollable crying, laughing, or both.
Trusted 100 mcg ventolin. Asthma Part 1.
Other transabdominal approaches include low anterior resections asthma treatment homeopathic cheap ventolin 100 mcg amex, total mesorectal excisions refractory asthma definition order 100mcg ventolin visa, and abdominal perineal resections asthma definition 666 generic 100 mcg ventolin free shipping. The Swedish Rectal Cancer Trial demonstrated an overall survival advantage to preoperative radiation asthma definition 8 ball discount 100 mcg ventolin with mastercard. The German Rectal Cancer Study Group investigated preoperative chemoradiation compared with postoperative therapy. Preoperative chemoradiation showed decreased local recurrence rates and improved sphincter function. External beam photon radiation therapy is utilized in the neoadjuvant, adjuvant, palliative and medically inoperable settings. The rectum extends from the transitional zone of the dentate line to the sigmoid colon. Tumors extending below the peritoneal reflection are considered rectal, while more proximal tumors are considered colonic. Treatment of rectal cancer requires interdisciplinary interaction between the radiologist, gastroenterologist, colorectal surgeon, radiation oncologist, and medical oncologist. For individuals who have T2 primary and negative margins, postoperative chemoradiation is appropriate after transanal excision. More recent trials of preoperative chemoradiation have established that as the preferred approach. Preoperative therapy affords the opportunity for downstaging of the tumor, improved resectability, greater likelihood of sphincter preservation, and improved local control. Individuals who present with synchronous limited metastatic disease amenable to R0 resection may also be candidates for definitive postoperative chemoradiation. Individuals with isolated pelvic or anastomotic recurrence who have not received prior radiation may be appropriately treated with preoperative or postoperative chemoradiation with or without intraoperative external beam photon radiation therapy or with primary chemoradiation if deemed unresectable. External beam photon radiation therapy treatment techniques and schedules for the treatment of rectal cancer A. External beam photon radiation therapy, preoperative and postoperative Treatment technique typically involves the use of multiple fields to encompass the regional lymph nodes and primary tumor site. Various treatment techniques may be used to decrease complications, such as prone positioning, customized immobilization. For unresectable cancers or individuals who are medically inoperable, doses higher than 54 Gy may be appropriate. In the postoperative setting with negative margins, 54 Gy in 30 fractions may be appropriate. External beam photon radiation therapy, palliative In previously un-irradiated individuals with unresectable metastatic disease and symptomatic local disease or near obstructing primaries who have reasonable life expectancy, external beam photon radiation therapy may be appropriate. Overview In the United States, the incidence of skin cancers outnumbers all other cancers combined, and basal cell cancers are twice as common as squamous cell skin cancers. While the two types share many characteristics, risk factors for local recurrence and for regional or distant metastases differ somewhat. Both types tend to occur in skin exposed to sunlight, and share the head and neck region as the area having the greatest risk for recurrence. Both occur more frequently and be more aggressive in immunocompromised transplant patients. In general, it is the squamous cell cancers that tend to be more aggressive, with a greater propensity to metastasize or to recur locoregionally. Anatomic location plays a role in risk stratification and is broken down into: "L" areas (trunk and extremities, excluding pretibia, hands, feet, nail units, ankles); "M" areas (cheeks, forehead, scalp, neck, pretibial); "H" areas (mask areas of face, including central face, eyelids, eyebrows, periorbital skin, lips, chin, overlying mandible, preauricular and postauricular skin, temple, ears, genitalia, hands, feet). Factors identified as placing the patient at increased risk for recurrence for basal and squamous cell skin cancers are included in Table 1. Management Treatment should be customized, taking into account specific factors and also patient preferences. The primary goal is to completely remove the tumor and to maximize functional and cosmetic preservation. Radiation therapy may be selected when cosmetic or functional outcome with surgery is expected to be inferior. In very low risk, superficial cancers, topical agents may be sufficient and cautiously used. When surgery is utilized, margin assessment using Mohs micrographic technique should include examining vertical sections of the specimen to assess deep margin and stage/depth of invasion. Photon and/or electron beam techniques are medically necessary for the treatment of basal cell and squamous cell cancers of the skin for any of the following: a. Definitive treatment for a cancer in a cosmetically significant location in which surgery would be disfiguring b. Adequate surgical margins have not been achieved and further resection is not possible c. Definitive management of large cancers as an alternative to major resection requiring significant plastic repair d. Definitive, preoperative, or postoperative adjuvant therapy for a cancers at risk for local or regional recurrence due to perineural, lymphovascular invasion, and/or metastatic adenopathy f. Radiation therapy should not be used in genetic conditions which predispose to skin cancer, such as xeroderma pigmentosum or basal cell nevus syndrome. Radiation treatments should be avoided or only used with great caution in cases of connective tissue disorders 2. When brachytherapy is required for treatment of skin cancers, up to ten (10) sessions is considered medically necessary. Superficial or kilovoltage (kV) xray treatments with low energy (up to 250 kV) external beam devices are generally used for thinner lesions. The beam energy and hardness (filtration) dictate the maximum thickness of a lesion that may be treated with this technique. Higher-energy external electron beam teletherapy (4 megaelectron volt [MeV] and greater) is most commonly utilized to treat the majority of localized lesions. The use of appropriate energy and thickness of build-up bolus material is required, along with proper sizing of the treatment field to account for the electron beam penumbra. Photon external beam teletherapy is required in circumstances in which other beams of lower energy are inadequate to reach the target depth. In the great majority of cases, simple appositional Complex technique is required, accompanied by lead, cerrobend, or other beam-shaping cutouts applied in the path of the beam and/or on the skin surface to match the shape of the target lesion. In complicated cases, such as when regional adenopathy or perineural invasion is present, more complicated techniques may be medically necessary. Treatment schedules with photons and/or electrons should be matched to the clinical circumstance, including size and depth of the lesion, histology, cosmetic goal, and risk of damage to underlying structures. Radiation doses typically range from 35 Gy in fractions of 7 Gy over 5 days, to 66 Gy in 33 fractions of 2 Gy over six and one-half weeks. The margin around tumor is typically different for basal and squamous histologies and for technique used (electrons, photons, superficial radiation). When regional nodes are to be treated, the dose range is 54 Gy to 66 Gy at 2 Gy per fraction. When multiple skin cancers are present and to be treated with radiation therapy, they should be treated concurrently rather than sequentially. Overview Malignant melanoma is increasing in incidence in the United States at a rate more rapidly for men than any other malignancy, and more rapidly for women for all malignancies except lung cancer. The incidence may be even higher, skewed by under-reporting of superficial and in situ cases. Like the non-melanoma skin cancers, excess sun exposure poses an increased risk of developing it, along with skin type, positive personal or family history, and environmental factors. Yet it can also occur in persons without substantial sun exposure and in any ethnic group or any color of skin. Survival is strongly inversely correlated with degree/depth of invasion, and decreases 50% with lymph node involvement. Some cases of melanoma take an indolent course while others are biologically much more aggressive. There are specific genetic alterations in distinct clinical subtypes of melanoma, often correlated with degree of sun damage. Non-mucosal, non-cutaneous melanomas also occur, such as in the uveal tract, and represent distinct presentations. The natural history of cutaneous melanoma is one of local invasion, lymphatic metastases, and hematologic dissemination.
Kerchner asthma definition bias order 100 mcg ventolin otc, 2007: Distribution of impacts of natural disasters across income groups: A case study of New Orleans asthma symptoms pdf purchase ventolin 100mcg without prescription. Luber asthma symptoms only in winter discount 100 mcg ventolin, 2012: Integrating climate change adaptation into public health practice: Using adaptive management to increase adaptive capacity and build resilience asthma treatment laba order ventolin 100 mcg overnight delivery. Meyer-Arendt, 2011: Direct and indirect mortality in Florida during the 2004 hurricane season. Balbus, 2014: Primary Protection: Enhancing Health Care Resilience for a Changing Climate. Hastak, 2011: Impact of flood damaged critical infrastructure on communities and industries. Webb, 2008: A place-based model for understanding community resilience to natural disasters. Winn, 2012: Extreme weather events and the critical importance of anticipatory adaptation and organizational resilience in responding to impacts. Herweijer, 2009: Adaptation to Climate Change: Linking Disaster Risk Reduction and Insurance. Gill, 2011: Hospital admissions for asthma and acute bronchitis in El Paso, Texas: Do age, sex, and insurance status modify the effects of dust and low wind events? Rosenberg, 2009: Neither rain nor hail nor sleet nor snow: Provider perspectives on the challenges of weather for home and community care. Murray, 2014: Power outages, extreme events and health: A systematic review of the literature from 2011-2012. Hwang, 2011: Health impact in New York City during the Northeastern blackout of 2003. McGeehin, 2000: An outbreak of carbon monoxide poisoning after a major ice storm in Maine. Yip, 2012: A review of disaster-related carbon monoxide poisoning: Surveillance, epidemiology, and opportunities for prevention. Thoroughman, 2011: Carbon monoxide poisoning after an ice storm in Kentucky, 2009. Muntner, 2009: Missed dialysis sessions and hospitalization in hemodialysis patients after Hurricane Katrina. Gantner, 2007: Preparing racially and ethnically diverse communities for public health emergencies. Solomon, 2011: Six climate change-related events in the United States accounted for about $14 billion in lost lives and health costs. Dannenberg, 2005: Impact of 2003 power outages on public health and emergency response. Kalkut, 2005: Effects of the August 2003 blackout on the New York City healthcare delivery system: A lesson for disaster preparedness. Clair, 2006: Impact of citywide blackout on an urban emergency medical services system. White, 2007: Demand for poison control center services "surged" during the 2003 blackout. Forsberg, 2013: Factors increasing vulnerability to health effects before, during and after floods. Hammond, 2006: Ariborne mold and endotoxin concentrations in New Orleans, Louisiana, after flooding, October through November 2005. Mendell, 2010: Association of residential dampness and mold with respiratory tract infections and bronchitis: A meta-analysis. Mendell, 2007: Meta-analyses of the associations of respiratory health effects with dampness and mold in homes. Douwes, 2011: Respiratory and allergic health effects of dampness, mold, and dampness-related agents: A review of the epidemiologic evidence. Kiefer, 2006: Infrastructure interdependency and the creation of a normal disaster: the case of Hurricane Katrina and the City of New Orleans. Villarini, 2013: Flooding associated with predecessor rain events over the Midwest United States. Hsia, 2011: Impact of the Red River catastrophic flood on women giving birth in North Dakota, 19942000. Buekens, 2008: Exposure to Hurricane Katrina, post-traumatic stress disorder and birth outcomes. Stough, 2010: Children with disabilities in the context of disaster: A social vulnerability perspective. Galarneau, 2012: Moisture transport into midlatitudes ahead of recurving tropical cyclones and its relevance in two predecessor rain events. Wade, 2009: A review of the potential impacts of climate change on surface water quality. Memberg, 2005: Impact of Anthropogenic Development on Coastal Ground-Water Hydrology in Southeastern Florida, 19002000. Langevin, 2005: Movement of the Saltwater Interface in the Surficial Aquifer System in Response to Hydrologic Stresses and Water-Management Practices, Broward County, Florida. Hoback, 2011: Water quality parameters of a Nebraska reservoir differ between drought and normal conditions. Thomas, 2009: Impacts of climate change on surface water quality in relation to drinking water production. Zwolsman, 2008: Impact of summer droughts on the water quality of the Meuse river. Wilson, 2004: Vulnerability of waterborne diseases to climate change in Canada: A review. Journal of Toxicology and Environmental Health, Part A: Current Issues, 67, 1667-1677. French, 2013: Potential effects of global environmental changes on cryptosporidiosis and giardiasis transmission. Sukhdeo, 2013: Drought-induced amplification of local and regional West Nile virus infection rates in New Jersey. Chase, 2007: Inter-annual associations between precipitation and human incidence of West Nile Virus in the United States. Torres-Pйrez, 2010: Hantaviruses in the Americas and their role as emerging pathogens. Van Ranst, 2009: Relating increasing hantavirus incidences to the changing climate: the mast connection. Mills, 2007: Brush mouse (Peromyscus boylii) population dynamics and hantavirus infection during a warm, drought period in southern Arizona. Wildlife and Emerging Zoonotic Diseases: the Biology, Circumstances and Consequences of Cross-Species Transmission. Panos, 2014: Epidemiology of Hantavirus infections in humans: A comprehensive, global overview. Yin, 2014: Changes in weather and climate extremes: State of knowledge relevant to air and water quality in the United States. Okin, 2011: Responses of wind erosion to climate-induced vegetation changes on the Colorado Plateau. Prather, 2012: Toward a more physical representation of precipitation scavenging in global chemistry models: Cloud overlap and ice physics and their impact on tropospheric ozone. Wiseman, 2008: Hope, despair and transformation: Climate change and the promotion of mental health and wellbeing. Urban, 2011: Regional and climatic controls on seasonal dust deposition in the southwestern U. Etzel, 1994: Surveillance for dust storms and respiratory diseases in Washington State, 1991. Mulkey, 2013: Multilobar lung infiltrates after exposure to dust storm: the haboob lung syndrome. Haberlie, 2015: Driving blind: Weather-related vision hazards and fatal motor vehicle crashes. Black, 2008: Fatalities associated with nonconvective high-wind events in the United States.
